DUMM binds irreversibly to insect nicotinic receptors and mimics the effects of acetylcholine, resulting in continuous nerve stimulation, incoordination, tremors, and death of the insect.

MARTIN SUPER inhibits chitin formation there by causing abnormal endocuticular deposition & abortive molting. As a result of chitin deficiency the procuticle of the insects loses its elasticity & the insect is unable to complete the molting process which leads to death of insect.

RETIRE is antagonist to the nicotinic acetyl choline receptor in the central nervous system. It disturbs the proper signal transmission system leading to excitation of nerve cell. Consequently a disorder of the nervous system occurs leading finally to the death of the treated insect.

CARBOMAX exerts its toxicity by reversibly inhibiting acetylcholinesterase leading to the persistent action of the otherwise hydrolyzed neurotransmitter, acetylcholine, on its postsynaptic receptors.

FILLER KING acts by interfering with the neuroendocrine system controlling ecdysone and juvenile hormone synthesis. It also inhibits the release of ecdysone from the hormoneproducing gland of insects resulting in moulting inhibition thereby antifeedant action is seen. In addition, FILLER KING causes inhibition of chitin synthesis.

CRIPPLE is a combination insecticide, in which thiamethoxam interferes with a specific receptor site in the insect’s nervous system. Once insects feed on the plant or come into contact with thiamethoxam, feeding is irreversibly stopped and insect damage halts whereas, lambda cyhalothrin acts on tiny chan nels through which sodium is pumped to cause excitation of neurons. They prevent the sodium channels from closing, resulting in continual nerve impulse transmission, tremors, and eventually, death.

SUPER CENSOR kills insects when they eat it or come in contact with it. It works by disrupting the normal function of the central nervous system in insects.

PRICE is a systemic insecticide with translaminar activity in plants. It has ovicidal and larvicidal activity against hemiptera, Coleoptera and Lepidoptera. Due to its ovicidal activity its gives longer duration of control.

TAICHI exhibits larvicidal activity as an orally ingested toxicant by targeting and disrupting the Ca2+ balance. This results in rapid cessation of feeding and extended residual control.

COANDA belongs to nicotinic acetylcholine receptor (nAChR) channel blockers and has systemic insecticide action with stomach and contact action.