DROLL affects the central and peripheral nervous system by interfering with sodium channel gating. It delays the closure of the sodium channel. It tends to hold the channel open for shorter times

DHOOMTARA interferes with a specific receptor site in the insect’s nervous system. Once insects feed on the plant or come into contact with thiamethoxam, feeding is irreversibly stopped and insect damage halts.

DEMET is an acetylcholinesterase inhibitor which disables cholinesterase, an enzyme essential for central nervous system function. It acts both by contact and through ingestion.

DANGER-25 acts by disrupting of VGSC function. Disruption of sodium channel function is mediated by specific binding sites. DANGER-25 binds with and disrupts the correct functioning of ion channel leading to death of insects.

CRIPPLE is a combination insecticide, in which thiamethoxam interferes with a specific receptor site in the insect’s nervous system. Once insects feed on the plant or come into contact with thiamethoxam, feeding is irreversibly stopped and insect damage halts whereas, lambda cyhalothrin acts on tiny chan nels through which sodium is pumped to cause excitation of neurons. They prevent the sodium channels from closing, resulting in continual nerve impulse transmission, tremors, and eventually, death.

COANDA belongs to nicotinic acetylcholine receptor (nAChR) channel blockers and has systemic insecticide action with stomach and contact action.

CENSOR XTRA kills insects when they eat it or come in contact with it. It works by disrupting the normal function of the central nervous system in insects.

CARBOMAX exerts its toxicity by reversibly inhibiting acetylcholinesterase leading to the persistent action of the otherwise hydrolyzed neurotransmitter, acetylcholine, on its postsynaptic receptors.